By Hermann Stefan and William H. Theodore (Eds.)
Epilepsy, half I, Basic ideas and Diagnosis, quantity 107, in the Handbook of medical Neurology series deals a finished assessment of our wisdom of the sector this day, together with epidemiology, easy mechanisms, animal versions, and subject matters of accelerating curiosity akin to the position of irritation in epilepsy. It offers a entire method of description of the medical, electrographic and imaging points of the epilepsies, with a transparent define of latest type and the position of contemporary diagnostic suggestions, in addition to neuropsychological and psychiatric elements of epilepsy. Chapters are authored via the world over revered neurologists with different views insuring intensity to the content material. Epilepsy, half II, Treatment, quantity 108, keeps assurance of epilepsy with a spotlight on therapy. The volumes should be a crucial source for uncomplicated scientists, scientific investigators, and all overall healthiness execs treating sufferers with epilepsy.
*A quantity within the guide of scientific Neurology sequence, which has an exceptional recognition because the world's such a lot entire resource of knowledge in neurology.
*International record of individuals together with the best employees within the field.
*Describes the advances that have happened in scientific neurology and the neurosciences, their impression at the figuring out of neurological problems and on sufferer care
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Extra info for Epilepsy
2009). Dravet syndrome or genetic (generalized) epilepsy with febrile seizures plus? Brain Dev 31: 394–400. CELLULAR BASES OF FOCAL AND GENERALIZED EPILEPSIES Schwartzkroin PA, Walsh CA (2000). Cortical malformations and epilepsy. Ment Retard Dev Disabil Res Rev 6: 268–280. Scimemi A, Semyanov A, Sperk G et al. (2005). Multiple and plastic receptors mediate tonic GABA(A)receptor currents in the hippocampus. J Neurosci 25: 10016–10024. Simonato M, Romualdi P (1996). Dynorphin and epilepsy. Prog Neurobiol 50: 557–583.
SCHWARTZKROIN CONCLUDING COMMENT The underlying cellular bases of seizures, both partial and generalized, are complex. Even in those cases where a single gene mutation “causes” the epilepsy, the cascade of mechanisms that lead to seizure activity inevitably involves many different processes. For the most part, the cellular mechanisms associated with seizure activity are variations on a “normal” theme. Thus, an understanding of seizure mechanisms must start with a detailed view of normal brain function.
The process of removing synaptically released glutamate from the extracellular space). , 2009). Under some conditions (high levels of activity, such as during seizures), this transport process may run in the reverse direction, resulting in glial release of glutamate into the extracellular space – a process that would then presumably further excite nearby neurons (Haydon and Carmignoto, 2006; Malarkey and Parpura, 2008). , 2008). Indeed, when these astrocytic metabotropic glutamate receptors are activated, one can see (using calcium imaging techniques) the spread of calcium “waves” through the glial network.