AIDS and Heart Disease by Ronald R. Watson

By Ronald R. Watson

The 1st connection with gather the most recent study and studies at the therapy and pathogenesis of center ailment in AIDS sufferers, this reference makes an attempt to enhance the analysis, administration, and treatment of HIV-associated middle failure via learning the lengthy results of immunosuppression and opportunistic and viral an infection, in addition to cofactors affecting middle illness comparable to drug-related cardiotoxicity, cocaine abuse, and dietary deficiencies on cardiac functionality.

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Galle et al. showed that both oxidized low-density lipoproteins (oxLDL) and oxidized lipoprotein (a) [oxLp (a)] increased the rate of apoptosis in endothelial cells and in rabbit aorta (84,85). This occurred in a dose-dependent manner (84,85). Oxidized lipoprotein-mediated induction of apoptosis was enhanced by inhibition of superoxide dismutase (SOD) (a scavenger of reactive oxygen species) and antioxidants, suggesting a role for oxidative stress in this process. Apoptotic endothelial cells can predispose to thrombus formation.

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